Title : Sustained c-Jun-NH2-kinase activity promotes epithelial-mesenchymal transition, invasion, and survival of breast cancer cells by regulating extracellular signal-regulated kinase activation.

Pub. Date : 2010 Feb

PMID : 20145041






1 Functional Relationships(s)
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1 In addition, hyperactive JNK attenuated the apoptosis of breast cancer cells treated by the chemotherapy drug paclitaxel, which is in contrast to the requirement for inducible JNK activity in response to cytotoxic chemotherapy. Paclitaxel mitogen-activated protein kinase 8 Homo sapiens