Title : FKBP38 protects Bcl-2 from caspase-dependent degradation.

Pub. Date : 2010 Mar 26

PMID : 20139069






2 Functional Relationships(s)
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1 We showed that the overexpression of FKBP38 attenuates reduction rate of Bcl-2, thus resulting in an increment of the intracellular Bcl-2 level, contributing to the resistance of apoptotic cell death induced by the treatment of kinetin riboside, an anticancer drug. kinetin riboside BCL2 apoptosis regulator Homo sapiens
2 We showed that the overexpression of FKBP38 attenuates reduction rate of Bcl-2, thus resulting in an increment of the intracellular Bcl-2 level, contributing to the resistance of apoptotic cell death induced by the treatment of kinetin riboside, an anticancer drug. kinetin riboside BCL2 apoptosis regulator Homo sapiens