Title : Preferentially increased nitration of alpha-synuclein at tyrosine-39 in a cellular oxidative model of Parkinson's disease.

Pub. Date : 2009 Sep 15

PMID : 19697948






2 Functional Relationships(s)
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1 Increased nitration of Tyr-39 on endogenous alpha-synuclein via elevations in MAO-B levels could be abrogated by the addition of deprenyl, a specific MAO-B inhibitor. Tyrosine monoamine oxidase B Homo sapiens
2 Increased nitration of Tyr-39 on endogenous alpha-synuclein via elevations in MAO-B levels could be abrogated by the addition of deprenyl, a specific MAO-B inhibitor. Tyrosine monoamine oxidase B Homo sapiens