Title : Oxidative stress decreases phosphatidylinositol 4,5-bisphosphate levels by deactivating phosphatidylinositol- 4-phosphate 5-kinase beta in a Syk-dependent manner.

Pub. Date : 2009 Aug 28

PMID : 19553680






1 Functional Relationships(s)
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1 These results suggest that during oxidative stress, as modeled by H(2)O(2) treatment, Syk-dependent tyrosine phosphorylation of PIP5Kbeta is the dominant post-translational modification that is responsible for the decrease in cellular PIP(2). Hydrogen Peroxide spleen associated tyrosine kinase Homo sapiens