Title : Chronic exposure to fibrin and fibrinogen differentially regulates intracellular Ca2+ in human pulmonary arterial smooth muscle and endothelial cells.

Pub. Date : 2009 Jun

PMID : 19363122






2 Functional Relationships(s)
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1 Furthermore, chronic exposure to thrombin, an activator of PAR, significantly reduced the peak agonist-induced Ca(2+) release in PAEC, but increased it in PASMC. pasmc coagulation factor II, thrombin Homo sapiens
2 In conclusion, chronic exposure to fibrinogen, fibrin, and thrombin caused differential changes in [Ca(2+)](cyt) in PAEC and PASMC. pasmc coagulation factor II, thrombin Homo sapiens