Title : Modulation of astrocytic metabolic phenotype by proinflammatory cytokines.

Pub. Date : 2008 Jul

PMID : 18383346






1 Functional Relationships(s)
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1 As far as mechanisms for oxidative stress defence are concerned, we observed that treatment with IL-1beta+TNFalpha decreases cellular glutathione content and increases glutathione release into the extracellular space while stimulating superoxide anion and nitric oxide production as well as H(2)O(2) release. Hydrogen Peroxide interleukin 1 beta Mus musculus