Title : Inflammation in pregnancy: its roles in reproductive physiology, obstetrical complications, and fetal injury.

Pub. Date : 2007 Dec

PMID : 18240548






2 Functional Relationships(s)
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1 The onset of premature labor in the context of infection is mediated by pro-inflammatory cytokines, such as interleukin (IL)-1beta and tumor necrosis factor alpha (TNF-alpha), as these cytokines are produced by intrauterine tissues in response to microbial products, can stimulate prostaglandin production, and induce labor in animals. Prostaglandins tumor necrosis factor Homo sapiens
2 The onset of premature labor in the context of infection is mediated by pro-inflammatory cytokines, such as interleukin (IL)-1beta and tumor necrosis factor alpha (TNF-alpha), as these cytokines are produced by intrauterine tissues in response to microbial products, can stimulate prostaglandin production, and induce labor in animals. Prostaglandins tumor necrosis factor Homo sapiens