Title : Drosophila brakeless interacts with atrophin and is required for tailless-mediated transcriptional repression in early embryos.

Pub. Date : 2007 Jun

PMID : 17503969






2 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 We discuss the possibility that human Brakeless homologs may influence the toxicity of polyglutamine-expanded Atrophin-1, which causes the human neurodegenerative disease dentatorubral-pallidoluysian atrophy (DRPLA). polyglutamine atrophin 1 Homo sapiens
2 We discuss the possibility that human Brakeless homologs may influence the toxicity of polyglutamine-expanded Atrophin-1, which causes the human neurodegenerative disease dentatorubral-pallidoluysian atrophy (DRPLA). polyglutamine atrophin 1 Homo sapiens