Title : Semaphorin 7A plays a critical role in TGF-beta1-induced pulmonary fibrosis.

Pub. Date : 2007 May 14

PMID : 17485510






4 Functional Relationships(s)
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1 TGF-beta(1) and bleomycin also activated phosphatidylinositol 3-kinase (PI3K) and protein kinase B (PKB)/AKT via SEMA 7A-dependent mechanisms, and PKB/AKT inhibition diminished TGF-beta(1)-induced fibrosis. Bleomycin thymoma viral proto-oncogene 1 Mus musculus
2 TGF-beta(1) and bleomycin also activated phosphatidylinositol 3-kinase (PI3K) and protein kinase B (PKB)/AKT via SEMA 7A-dependent mechanisms, and PKB/AKT inhibition diminished TGF-beta(1)-induced fibrosis. Bleomycin thymoma viral proto-oncogene 1 Mus musculus
3 TGF-beta(1) and bleomycin also activated phosphatidylinositol 3-kinase (PI3K) and protein kinase B (PKB)/AKT via SEMA 7A-dependent mechanisms, and PKB/AKT inhibition diminished TGF-beta(1)-induced fibrosis. Bleomycin thymoma viral proto-oncogene 1 Mus musculus
4 TGF-beta(1) and bleomycin also activated phosphatidylinositol 3-kinase (PI3K) and protein kinase B (PKB)/AKT via SEMA 7A-dependent mechanisms, and PKB/AKT inhibition diminished TGF-beta(1)-induced fibrosis. Bleomycin thymoma viral proto-oncogene 1 Mus musculus