Pub. Date : 2007 May
PMID : 17299072
6 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | CONTEXT: Retention of technetium-(99m)-sestamibi ((99m)Tc-sestamibi) by parathyroid adenomas appears to be due to the loss of at least one membrane transporter, multidrug resistance 1 (MDR1), and possibly another, multidrug resistance-associated protein 1 (MRP1). | Technetium Tc 99m Sestamibi | ATP binding cassette subfamily B member 1 | Homo sapiens |
2 | CONTEXT: Retention of technetium-(99m)-sestamibi ((99m)Tc-sestamibi) by parathyroid adenomas appears to be due to the loss of at least one membrane transporter, multidrug resistance 1 (MDR1), and possibly another, multidrug resistance-associated protein 1 (MRP1). | Technetium Tc 99m Sestamibi | ATP binding cassette subfamily B member 1 | Homo sapiens |
3 | CONTEXT: Retention of technetium-(99m)-sestamibi ((99m)Tc-sestamibi) by parathyroid adenomas appears to be due to the loss of at least one membrane transporter, multidrug resistance 1 (MDR1), and possibly another, multidrug resistance-associated protein 1 (MRP1). | Technetium Tc 99m Sestamibi | ATP binding cassette subfamily B member 1 | Homo sapiens |
4 | CONTEXT: Retention of technetium-(99m)-sestamibi ((99m)Tc-sestamibi) by parathyroid adenomas appears to be due to the loss of at least one membrane transporter, multidrug resistance 1 (MDR1), and possibly another, multidrug resistance-associated protein 1 (MRP1). | Technetium Tc 99m Sestamibi | ATP binding cassette subfamily B member 1 | Homo sapiens |
5 | Among all glands, hypermethylation for MDR1 was more likely in (99m)Tc-sestamibi-positive scans (P < 0.001). | Technetium Tc 99m Sestamibi | ATP binding cassette subfamily B member 1 | Homo sapiens |
6 | CONCLUSION: In parathyroid tissue, hypermethylation of the MDR1 gene decreases its expression and is associated with increased detection of parathyroid adenomas by (99m)Tc-sestamibi parathyroid scans. | Technetium Tc 99m Sestamibi | ATP binding cassette subfamily B member 1 | Homo sapiens |