Title : Molecular mechanisms of RET receptor-mediated oncogenesis in multiple endocrine neoplasia 2B.

Pub. Date : 2006 Nov 15

PMID : 17108110






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 The disease is caused primarily by a methionine to threonine substitution of residue 918 in the kinase domain of the RET receptor (2B-RET); however, the molecular mechanisms that lead to the disease phenotype are unclear. Methionine ret proto-oncogene Homo sapiens
2 The disease is caused primarily by a methionine to threonine substitution of residue 918 in the kinase domain of the RET receptor (2B-RET); however, the molecular mechanisms that lead to the disease phenotype are unclear. Methionine ret proto-oncogene Homo sapiens