Title : CA150 expression delays striatal cell death in overexpression and knock-in conditions for mutant huntingtin neurotoxicity.

Pub. Date : 2006 Apr 26

PMID : 16641246






4 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Transcriptional dysregulation caused by expanded polyglutamines (polyGlns) in huntingtin (htt) may be central to cell-autonomous mechanisms for neuronal cell death in Huntington"s disease (HD) pathogenesis. polyglutamine huntingtin Rattus norvegicus
2 Transcriptional dysregulation caused by expanded polyglutamines (polyGlns) in huntingtin (htt) may be central to cell-autonomous mechanisms for neuronal cell death in Huntington"s disease (HD) pathogenesis. polyglutamine huntingtin Rattus norvegicus
3 Transcriptional dysregulation caused by expanded polyglutamines (polyGlns) in huntingtin (htt) may be central to cell-autonomous mechanisms for neuronal cell death in Huntington"s disease (HD) pathogenesis. polyglutamine huntingtin Rattus norvegicus
4 Transcriptional dysregulation caused by expanded polyglutamines (polyGlns) in huntingtin (htt) may be central to cell-autonomous mechanisms for neuronal cell death in Huntington"s disease (HD) pathogenesis. polyglutamine huntingtin Rattus norvegicus