Title : Nontranscriptional regulation of cardiac repolarization currents by testosterone.

Pub. Date : 2005 Sep 20

PMID : 16157773






7 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 A nitric oxide (NO) scavenger and an inhibitor of NO synthase 3 (NOS3) reversed the effects of testosterone on APD, which suggests that NO released from NOS3 is responsible for the electrophysiological effects of testosterone. Testosterone nitric oxide synthase 3 Homo sapiens
2 A nitric oxide (NO) scavenger and an inhibitor of NO synthase 3 (NOS3) reversed the effects of testosterone on APD, which suggests that NO released from NOS3 is responsible for the electrophysiological effects of testosterone. Testosterone nitric oxide synthase 3 Homo sapiens
3 A nitric oxide (NO) scavenger and an inhibitor of NO synthase 3 (NOS3) reversed the effects of testosterone on APD, which suggests that NO released from NOS3 is responsible for the electrophysiological effects of testosterone. Testosterone nitric oxide synthase 3 Homo sapiens
4 A nitric oxide (NO) scavenger and an inhibitor of NO synthase 3 (NOS3) reversed the effects of testosterone on APD, which suggests that NO released from NOS3 is responsible for the electrophysiological effects of testosterone. Testosterone nitric oxide synthase 3 Homo sapiens
5 A nitric oxide (NO) scavenger and an inhibitor of NO synthase 3 (NOS3) reversed the effects of testosterone on APD, which suggests that NO released from NOS3 is responsible for the electrophysiological effects of testosterone. Testosterone nitric oxide synthase 3 Homo sapiens
6 A nitric oxide (NO) scavenger and an inhibitor of NO synthase 3 (NOS3) reversed the effects of testosterone on APD, which suggests that NO released from NOS3 is responsible for the electrophysiological effects of testosterone. Testosterone nitric oxide synthase 3 Homo sapiens
7 Immunoblot analysis revealed that testosterone induced phosphorylation of Akt and NOS3. Testosterone nitric oxide synthase 3 Homo sapiens