Title : Mechanism and effect of thrombospondin-4 polymorphisms on neutrophil function.

Pub. Date : 2005 Dec 1

PMID : 16099885






2 Functional Relationships(s)
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1 Despite the similarity in these responses, the P387 variant induced more robust tyrosine phosphorylation of the stress-related mitogen-activated protein kinases (MAPKs): p38MAPK and c-Jun NH2-terminal kinase (JNK), as well as signal transducer and activator of transcription-1 (STAT1) and heat shock protein 27 (HSP27) than the A387 variant. Tyrosine mitogen-activated protein kinase 8 Homo sapiens
2 Despite the similarity in these responses, the P387 variant induced more robust tyrosine phosphorylation of the stress-related mitogen-activated protein kinases (MAPKs): p38MAPK and c-Jun NH2-terminal kinase (JNK), as well as signal transducer and activator of transcription-1 (STAT1) and heat shock protein 27 (HSP27) than the A387 variant. Tyrosine mitogen-activated protein kinase 8 Homo sapiens