Title : Tumor necrosis factor-alpha mediates the proliferation of rat C6 glioma cells via beta-adrenergic receptors.

Pub. Date : 2005 Sep

PMID : 16005083






1 Functional Relationships(s)
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1 In the present study, we observed that isoproterenol, a beta-adrenergic receptor (beta-AR) agonist, stimulated rat C6 glioma cell proliferation, while propranolol, a beta-AR blocker, greatly reduced the proliferative effect of TNF-alpha on C6 cells. Propranolol tumor necrosis factor Rattus norvegicus