Title : CCK enhances response to gastric distension by acting on capsaicin-insensitive vagal afferents.

Pub. Date : 2005 Sep

PMID : 15905220






3 Functional Relationships(s)
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1 Capsaicin treatment destroys vagal afferent C fibers and markedly attenuates reduction of food intake and induction of hindbrain Fos expression by CCK. Capsaicin Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus
2 To test this hypothesis, we quantified expression of Fos-like immunoreactivity (Fos) in the dorsal vagal complex (DVC) of capsaicin-treated (Cap) and control rats (Veh), following gastric balloon distension alone and in combination with CCK injection. Capsaicin Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus
3 To test this hypothesis, we quantified expression of Fos-like immunoreactivity (Fos) in the dorsal vagal complex (DVC) of capsaicin-treated (Cap) and control rats (Veh), following gastric balloon distension alone and in combination with CCK injection. Capsaicin Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus