Title : Striatal damage and oxidative stress induced by the mitochondrial toxin malonate are reduced in clorgyline-treated rats and MAO-A deficient mice.

Pub. Date : 2004 Apr

PMID : 15098936






4 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Intrastriatal administration of the succinate dehydrogenase (SDH) inhibitor malonate produces neuronal injury by a "secondary excitotoxic" mechanism involving the generation of reactive oxygen species (ROS). Reactive Oxygen Species aminoadipate-semialdehyde synthase Mus musculus
2 Intrastriatal administration of the succinate dehydrogenase (SDH) inhibitor malonate produces neuronal injury by a "secondary excitotoxic" mechanism involving the generation of reactive oxygen species (ROS). Reactive Oxygen Species aminoadipate-semialdehyde synthase Mus musculus
3 Intrastriatal administration of the succinate dehydrogenase (SDH) inhibitor malonate produces neuronal injury by a "secondary excitotoxic" mechanism involving the generation of reactive oxygen species (ROS). Reactive Oxygen Species aminoadipate-semialdehyde synthase Mus musculus
4 Intrastriatal administration of the succinate dehydrogenase (SDH) inhibitor malonate produces neuronal injury by a "secondary excitotoxic" mechanism involving the generation of reactive oxygen species (ROS). Reactive Oxygen Species aminoadipate-semialdehyde synthase Mus musculus