Title : Growth inhibition by anchorage-deficiency is associated with increased level but reduced phosphorylation of mutant p53.

Pub. Date : 1992 Apr

PMID : 1506270






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Immunoprecipitation analysis revealed that mutant p53 was phosphorylated at tyrosine in the anchorage-provided cells. Tyrosine tumor protein p53 Homo sapiens
2 These results demonstrated that the growth inhibition by anchorage-deficiency or by herbimycin A is associated with an elevated p53 level and reduced p53 phosphorylation at tyrosine. Tyrosine tumor protein p53 Homo sapiens