Title : Background potassium channel block and TRPV1 activation contribute to proton depolarization of sensory neurons from humans with neuropathic pain.

Pub. Date : 2004 Mar

PMID : 15016092






1 Functional Relationships(s)
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1 Consistent with the involvement of TRPV1, these proton-evoked currents were blocked by capsazepine and were only found in neurons that responded to capsaicin with an increase in membrane conductance. Capsaicin transient receptor potential cation channel subfamily V member 1 Homo sapiens