Title : Adenovirus-mediated delivery and expression of a cAMP-dependent protein kinase inhibitor gene to BEAS-2B epithelial cells abolishes the anti-inflammatory effects of rolipram, salbutamol, and prostaglandin E2: a comparison with H-89.

Pub. Date : 2004 May

PMID : 14747610






1 Functional Relationships(s)
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1 Paradoxically, H-89 antagonized MCP-1-induced [(3)H]AA release and enhanced the inhibitory effect of submaximal concentrations of rolipram and 8-bromo-cAMP. N-(2-(4-bromocinnamylamino)ethyl)-5-isoquinolinesulfonamide C-C motif chemokine ligand 2 Homo sapiens