Title : Heme oxygenase-1 and its reaction product, carbon monoxide, prevent inflammation-related apoptotic liver damage in mice.

Pub. Date : 2003 Oct

PMID : 14512878






1 Functional Relationships(s)
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1 In addition, overexpression of HO-1 by adenoviral gene transfer resulted in protection from apoptotic liver injury, whereas inhibition of HO-1 enzymatic activity by tin-protoporphyrin-IX (SnPP) abrogated the protective effect. tin protoporphyrin IX heme oxygenase 1 Mus musculus