Title : Heme oxygenase-1 and its reaction product, carbon monoxide, prevent inflammation-related apoptotic liver damage in mice.

Pub. Date : 2003 Oct

PMID : 14512878






1 Functional Relationships(s)
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1 Exogenous CO administration or treatment with the CO-releasing agent methylene chloride mimicked the protective effect of HO-1, whereas treatment with exogenous biliverdin or overexpression of ferritin by recombinant adenoviral gene transfer did not. Methylene Chloride heme oxygenase 1 Mus musculus