Title : The biology and pathobiology of the ECL cells.

Pub. Date : 1992 Nov-Dec

PMID : 1341077






2 Functional Relationships(s)
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1 Recently, we could show that alpha-fluoromethylhistidine, which is a suicide inhibitor of histidine decarboxylase, effectively depletes the ECL cells of histamine and that the histamine-depleted ECL cells respond to gastrin with hyperplasia in a manner identical to normal ECL cells. Histamine histidine decarboxylase Rattus norvegicus
2 Recently, we could show that alpha-fluoromethylhistidine, which is a suicide inhibitor of histidine decarboxylase, effectively depletes the ECL cells of histamine and that the histamine-depleted ECL cells respond to gastrin with hyperplasia in a manner identical to normal ECL cells. Histamine histidine decarboxylase Rattus norvegicus