Title : Inhibition by terbutaline of nitric oxide and superoxide anion levels of endotoxin-induced organs injury in the anesthetized rat.

Pub. Date : 2003 Mar

PMID : 12630530






2 Functional Relationships(s)
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1 Overproduction of TNF-alpha and NO* (produced by inducible NO synthase [iNOS] examined by Western blot analysis in the lung and the liver) is inhibited by treatment of LPS rats with terbutaline. Terbutaline tumor necrosis factor Rattus norvegicus
2 These findings suggest that the amelioration of circulatory failure and organs injury by terbutaline is associated with its suppression in TNF-alpha, O2*- and NO (via iNOS) production in animals with endotoxic shock. Terbutaline tumor necrosis factor Rattus norvegicus