Title : Paclitaxel induces prolonged activation of the Ras/MEK/ERK pathway independently of activating the programmed cell death machinery.

Pub. Date : 2001 Jun 1

PMID : 11278851






4 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Paclitaxel induces prolonged activation of the Ras/MEK/ERK pathway independently of activating the programmed cell death machinery. Paclitaxel mitogen-activated protein kinase 1 Homo sapiens
2 All three mitogen-activated protein kinase (MAPK) family members, c-Jun N-terminal kinase (JNK), p38 MAPK, and extracellular signal-regulated kinase (ERK) were activated upon paclitaxel treatment. Paclitaxel mitogen-activated protein kinase 1 Homo sapiens
3 All three mitogen-activated protein kinase (MAPK) family members, c-Jun N-terminal kinase (JNK), p38 MAPK, and extracellular signal-regulated kinase (ERK) were activated upon paclitaxel treatment. Paclitaxel mitogen-activated protein kinase 1 Homo sapiens
4 All three mitogen-activated protein kinase (MAPK) family members, c-Jun N-terminal kinase (JNK), p38 MAPK, and extracellular signal-regulated kinase (ERK) were activated upon paclitaxel treatment. Paclitaxel mitogen-activated protein kinase 1 Homo sapiens