Title : The delayed induction of c-jun in apoptotic human leukemic lymphoblasts is primarily transcriptional.

Pub. Date : 2000 Dec 15

PMID : 11226825






3 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Use of cycloheximide to block protein synthesis strongly induced c-jun mRNA, suggesting that there had been relief from a labile protein repressor of transcription. Cycloheximide Jun proto-oncogene, AP-1 transcription factor subunit Homo sapiens
2 The high induction by cycloheximide obscured simple interpretation of elevated c-jun mRNA levels after concomitant administration of cycloheximide and dexamethasone. Cycloheximide Jun proto-oncogene, AP-1 transcription factor subunit Homo sapiens
3 The high induction by cycloheximide obscured simple interpretation of elevated c-jun mRNA levels after concomitant administration of cycloheximide and dexamethasone. Cycloheximide Jun proto-oncogene, AP-1 transcription factor subunit Homo sapiens