Title : Intermediate syndrome secondary to ingestion of chlorpiriphos.

Pub. Date : 2001 Feb

PMID : 11205075






1 Functional Relationships(s)
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1 These clinical manifestations occur from liposoluble organophosphates or metabolites with long-lasting half time, causeing delayed inhibition of acetylcholinesterase and subsequent burn out of the neuromuscular junction from acetylcholine overstimulation. Organophosphates acetylcholinesterase (Cartwright blood group) Homo sapiens