Title : Allosteric modulation of nicotinic receptors as a treatment strategy for Alzheimer's disease.

Pub. Date : 2000 Sep

PMID : 10971047






4 Functional Relationships(s)
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1 The two most common approaches to correcting these cholinergic deficits are to increase the synaptic availability of acetylcholine (ACh) by inhibiting acetylcholinesterase (AChE), or to mimic the effects of ACh (nicotinic agonists) by acting directly on nicotinic receptors. Acetylcholine acetylcholinesterase (Cartwright blood group) Homo sapiens
2 The two most common approaches to correcting these cholinergic deficits are to increase the synaptic availability of acetylcholine (ACh) by inhibiting acetylcholinesterase (AChE), or to mimic the effects of ACh (nicotinic agonists) by acting directly on nicotinic receptors. Acetylcholine acetylcholinesterase (Cartwright blood group) Homo sapiens
3 The two most common approaches to correcting these cholinergic deficits are to increase the synaptic availability of acetylcholine (ACh) by inhibiting acetylcholinesterase (AChE), or to mimic the effects of ACh (nicotinic agonists) by acting directly on nicotinic receptors. Acetylcholine acetylcholinesterase (Cartwright blood group) Homo sapiens
4 The two most common approaches to correcting these cholinergic deficits are to increase the synaptic availability of acetylcholine (ACh) by inhibiting acetylcholinesterase (AChE), or to mimic the effects of ACh (nicotinic agonists) by acting directly on nicotinic receptors. Acetylcholine acetylcholinesterase (Cartwright blood group) Homo sapiens