Title : Endocrinological study of the dopaminergic regulation of prolactin release in metastatic breast cancer.

Pub. Date : 1999 Nov-Dec

PMID : 10774572






2 Functional Relationships(s)
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1 In addition, by showing a paradoxical rise of PRL in response to L-dopa, which inhibits PRL secretion in physiological conditions, this study would suggest that breast cancer-related hyperprolactinemia may depend at least in part on endogenous disease-related neuroendocrine alterations. Levodopa prolactin Homo sapiens
2 In addition, by showing a paradoxical rise of PRL in response to L-dopa, which inhibits PRL secretion in physiological conditions, this study would suggest that breast cancer-related hyperprolactinemia may depend at least in part on endogenous disease-related neuroendocrine alterations. Levodopa prolactin Homo sapiens