Title : Identification of amino acid residues and protein kinases involved in the regulation of NFATc subcellular localization.

Pub. Date : 2000 Feb 4

PMID : 10652349






1 Functional Relationships(s)
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1 Moreover, we found that overexpression of JNK, ERK, or p38 is able to block ionomycin-induced NFATc nuclear translocation, whereas treatment of cells with both PD98059 and SB202190, which inhibit MAPK/SAPK signaling pathways, is sufficient to trigger NFATc nuclear localization. Ionomycin mitogen-activated protein kinase 14 Homo sapiens