Pub. Date : 1999 Jun
PMID : 10475066
6 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | The Glu-298-->Asp (894G-->T) mutation at exon 7 of the endothelial nitric oxide synthase gene and coronary artery disease. | Glutamic Acid | nitric oxide synthase 3 | Homo sapiens |
| 2 | We examined associations between the endothelial nitric oxide synthase (eNOS) gene Glu-298-->Asp (894G-->T) mutation and the occurrence and severity of angiographically defined coronary artery disease (CAD). | Glutamic Acid | nitric oxide synthase 3 | Homo sapiens |
| 3 | We examined associations between the endothelial nitric oxide synthase (eNOS) gene Glu-298-->Asp (894G-->T) mutation and the occurrence and severity of angiographically defined coronary artery disease (CAD). | Glutamic Acid | nitric oxide synthase 3 | Homo sapiens |
| 4 | The newly identified eNOS Glu-298-->Asp mutation in exon 7 is common and likely to be functional. | Glutamic Acid | nitric oxide synthase 3 | Homo sapiens |
| 5 | We genotyped 763 white Australians undergoing coronary angiography for the eNOS Glu-298-->Asp mutation. | Glutamic Acid | nitric oxide synthase 3 | Homo sapiens |
| 6 | In conclusion, the eNOS Glu-298-->Asp mutation is common, occurring with an allele frequency of 32.5%, but is not associated with either the occurrence or severity of CAD in the Australian population or with other established coronary risk factors assessed in our study. | Glutamic Acid | nitric oxide synthase 3 | Homo sapiens |