Title : Angiotensin-converting enzyme inhibition and AT1 receptor blockade modify the pressure-natriuresis relationship by additive mechanisms in rats with human renin and angiotensinogen genes.

Pub. Date : 1999 Aug

PMID : 10446934






1 Functional Relationships(s)
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1 Therefore, submaximal ACE inhibition enhanced sodium excretion mainly by increasing RBF and GFR, whereas submaximal AT1 receptor blockade decreased tubular sodium and water reabsorption. Sodium angiotensin I converting enzyme Homo sapiens