Title : Effects of erythromycin on experimental extrinsic allergic alveolitis.

Pub. Date : 1999 Feb

PMID : 10051731






2 Functional Relationships(s)
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1 Histopathological studies revealed that erythromycin markedly decreased neutrophils in the lung and skin lesions and myeloperoxidase in the lung, simultaneously with inhibiting ICAM-1 expression. Erythromycin intercellular adhesion molecule 1 Homo sapiens
2 CONCLUSIONS: The therapeutic dosage of erythromycin significantly suppressed acute neutrophil influx into the lung, intradermal Arthus reaction and the expression of ICAM-1 in the lesions of experimental EAA. Erythromycin intercellular adhesion molecule 1 Homo sapiens