Title : Induced expression of p16(INK4a) inhibits both CDK4- and CDK2-associated kinase activity by reassortment of cyclin-CDK-inhibitor complexes.

Pub. Date : 1999 Mar

PMID : 10022885






4 Functional Relationships(s)
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1 To investigate the mode of action of the p16(INK4a) tumor suppressor protein, we have established U2-OS cells in which the expression of p16(INK4a) can be regulated by addition or removal of isopropyl-beta-D-thiogalactopyranoside. Isopropyl Thiogalactoside cyclin dependent kinase inhibitor 2A Homo sapiens
2 To investigate the mode of action of the p16(INK4a) tumor suppressor protein, we have established U2-OS cells in which the expression of p16(INK4a) can be regulated by addition or removal of isopropyl-beta-D-thiogalactopyranoside. Isopropyl Thiogalactoside cyclin dependent kinase inhibitor 2A Homo sapiens
3 To investigate the mode of action of the p16(INK4a) tumor suppressor protein, we have established U2-OS cells in which the expression of p16(INK4a) can be regulated by addition or removal of isopropyl-beta-D-thiogalactopyranoside. Isopropyl Thiogalactoside cyclin dependent kinase inhibitor 2A Homo sapiens
4 To investigate the mode of action of the p16(INK4a) tumor suppressor protein, we have established U2-OS cells in which the expression of p16(INK4a) can be regulated by addition or removal of isopropyl-beta-D-thiogalactopyranoside. Isopropyl Thiogalactoside cyclin dependent kinase inhibitor 2A Homo sapiens